Effects of adding P38 mitogen-activated protein-kinase inhibitor to celsior solution in canine heart transplantation from non???heart-beating donors
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Transplantation
Reference29 articles.
1. Increased Myocardial Tumor Necrosis Factor-α in a Crystalloid-Perfused Model of Cardiac Ischemia-Reperfusion Injury
2. Inhibition of Extracellular Signal–Regulated Kinase Enhances Ischemia/Reoxygenation–Induced Apoptosis in Cultured Cardiac Myocytes and Exaggerates Reperfusion Injury in Isolated Perfused Heart
3. “Stress-Responsive” Mitogen-Activated Protein Kinases (c-Jun N-Terminal Kinases and p38 Mitogen-Activated Protein Kinases) in the Myocardium
4. Stimulation of the Stress-Activated Mitogen-Activated Protein Kinase Subfamilies in Perfused Heart
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1. Effects of T3 Administration on Ex Vivo Rat Hearts Subjected to Normothermic Perfusion: Therapeutic Implications in Donor Heart Preservation and Repair;Transplant International;2023-02-07
2. PREVENTION AND TREATMENT OF ISCHEMIA-REPERFUSION SYNDROME;Nephrology (Saint-Petersburg);2019-02-21
3. Salidroside pretreatment attenuates apoptosis and autophagy during hepatic ischemia–reperfusion injury by inhibiting the mitogen-activated protein kinase pathway in mice;Drug Design, Development and Therapy;2017-07
4. Donation after circulatory death heart transplantation;Current Opinion in Organ Transplantation;2017-06
5. HEMO2life as a protective additive to Celsior solution for static storage of donor hearts prior to transplantation;Artificial Cells, Nanomedicine, and Biotechnology;2017-01-12
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