KCNQ1 G219E and TRPM4 T160M polymorphisms are involved in the pathogenesis of long QT syndrome-Reference-Cited by-同舟云学术

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KCNQ1 G219E and TRPM4 T160M polymorphisms are involved in the pathogenesis of long QT syndrome

Published:2021-01-15 Issue:2 Volume:100 Page:e24032
ISSN:0025-7974
Container-title:Medicine
language:en
Short-container-title:

Author:

Zhao Yang,Feng Min,Shang Lu-Xiang,Sun Hua-xin,Zhou Xian-Hui,Lu Yan-Mei,Zhang Ling,Xing Qiang,Li Yao-dong,Tang Bao-Peng

Funder

National Natural Science Foundation

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

General Medicine

Reference19 articles.

1. Long-QT syndrome: from genetics to management;Schwartz;Circ Arrhythm Electrophysiol,2012

2. Idiopathic long QT syndrome: progress and questions;Schwartz;Am Heart J,1985

3. Linkage of a cardiac arrhythmia, the long QT syndrome, and the Harvey ras-1 gene;Keating;Science,1991

4. SCN5A mutations associated with an inherited cardiac arrhythmia, long QT syndrome;Wang;Cell,1995

5. A molecular basis for cardiac arrhythmia: HERG mutations cause long QT syndrome;Curran;Cell,1995

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1. The Role of TRPM4 in Cardiac Electrophysiology and Arrhythmogenesis;International Journal of Molecular Sciences;2023-07-22

2. Pharmacological Modulation and (Patho)Physiological Roles of TRPM4 Channel—Part 2: TRPM4 in Health and Disease;Pharmaceuticals;2021-12-28

3. Electrophysiological Effects of the Transient Receptor Potential Melastatin 4 Channel Inhibitor (4-Chloro-2-(2-chlorophenoxy)acetamido) Benzoic Acid (CBA) in Canine Left Ventricular Cardiomyocytes;International Journal of Molecular Sciences;2021-08-31

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