Impact of V-ets Erythroblastosis Virus E26 Oncogene Homolog 1 Gene Polymorphisms Upon Susceptibility to Autoimmune Diseases-Reference-Cited by-同舟云学术

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Impact of V-ets Erythroblastosis Virus E26 Oncogene Homolog 1 Gene Polymorphisms Upon Susceptibility to Autoimmune Diseases

Published:2015-06 Issue:22 Volume:94 Page:e923
ISSN:0025-7974
Container-title:Medicine
language:en
Short-container-title:

Author:

Zhou Ye,Liu Miao,Li Jun,Hashmi Fiza,Mao Zhi,Zhang Ning,Zhou Liang,Lv Weiran,Zheng Jingwei,Nie Xiaoli,Li Changzheng

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

General Medicine

Reference28 articles.

1. Autoimmune disease: why and where it occurs.;Marrack;Nat Med,2001

2. Lymphocyte activation gene-3 (LAG-3) negatively regulates environmentally-induced autoimmunity.;Jha;PLoS One,2014

3. The immunogenetic architecture of autoimmune disease.;Goris;Cold Spring Harb Perspect Biol,2012

4. Environmental agents and autoimmune diseases.;Miller;Adv Exp Med Biol,2011

5. Increased expression of avian erythroblastosis virus E26 oncogene homolog 1 in World Health Organization grade 1 meningiomas is associated with an elevated risk of recurrence and is correlated with the expression of its target genes matrix metalloproteinase-2 and MMP-9.;Okuducu;Cancer,2011

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