Data-independent acquisition-based quantitative proteomic analysis of m.3243A>G MELAS reveals novel potential pathogenesis and therapeutic targets-Reference-Cited by-同舟云学术

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Data-independent acquisition-based quantitative proteomic analysis of m.3243A>G MELAS reveals novel potential pathogenesis and therapeutic targets

Published:2022-10-14 Issue:41 Volume:101 Page:e30938
ISSN:1536-5964
Container-title:Medicine
language:en
Short-container-title:

Author:

Chang Xueli,Yin Zhaoxu,Zhang Wei,Shi Jiaying,Pu Chuanqiang,Shi Qiang,Wang Juan,Zhang Jing,Yan Li,Yang Wenqu,Guo Junhong

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

General Medicine

Reference44 articles.

1. Prevalence of mitochondrial DNA disease in adults.;Schaefer;Ann Neurol,2008

2. Prevalence of nuclear and mitochondrial DNA mutations related to adult mitochondrial disease.;Gorman;Ann Neurol,2015

3. Mitochondrial myopathy, encephalopathy, lactic acidosis, and strokelike episodes: a distinctive clinical syndrome.;Pavlakis;Ann Neurol,1984

4. Clinical features of MELAS and its relation with A3243G gene point mutation.;Zhang;Int J Clin Exp Path,2015

5. Prevalence and natural history of heart disease in adults with primary mitochondrial respiratory chain disease.;Limongelli;Eur J Heart Fail,2010

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Multiomics analysis reveals serine catabolism as a potential therapeutic target for MELAS;The FASEB Journal;2024-06-12

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