Activation of the Nuclear Receptor Fxr Improves Intestinal Cell Tolerance to Ischemia–Reperfusion Injury-Reference-Cited by-同舟云学术

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Activation of the Nuclear Receptor Fxr Improves Intestinal Cell Tolerance to Ischemia–Reperfusion Injury

Published:2018-09 Issue:3 Volume:50 Page:316-323
ISSN:1073-2322
Container-title:Shock
language:en
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Author:

Wang Xuan,Li Shuo,Chen Man,Liu Jing,Dong Ruirui,Wang Huamin,Zhu Shigong

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Critical Care and Intensive Care Medicine,Emergency Medicine

Reference23 articles.

1. The effect of antioxidant supplementation on bacterial translocation after intestinal ischemia and reperfusion;Tassopoulos;Redox Rep,2017

2. Effects of lazaroid U-74389G on intestinal ischemia and reperfusion injury in porcine experimental model;Flessas;Int J Surg,2015

3. Restoration of barrier function in injured intestinal mucosa;Blikslager;Physiol Rev,2007

4. Intestinal ischemia/reperfusion: microcirculatory pathology and functional consequences;Vollmar;Langenbecks Arch Surg,2011

5. Ischemia-reperfusion injury: processes in pathogenetic networks: a review;de Groot;Transplant Proc,2007

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2. The role of intestinal microbiota and its metabolites in intestinal and extraintestinal organ injury induced by intestinal ischemia reperfusion injury;International Journal of Biological Sciences;2022

3. The Pathophysiology of Farnesoid X Receptor (FXR) in the GI Tract: Inflammation, Barrier Function and Innate Immunity;Cells;2021-11-17

4. Dioscin alleviates lung ischemia/reperfusion injury by regulating FXR-mediated oxidative stress, apoptosis, and inflammation;European Journal of Pharmacology;2021-10

5. Farnesoid X receptor promotes renal ischaemia‐reperfusion injury by inducing tubular epithelial cell apoptosis;Cell Proliferation;2021-02-16

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