Regulator of G protein signaling 5 is a determinant of gestational hypertension and preeclampsia

Author:

Holobotovskyy Vasyl1,Chong Yee Seng1,Burchell Jennifer1,He Bo1,Phillips Michael12,Leader Leo3,Murphy Timothy V.4,Sandow Shaun L.45,McKitrick Douglas J.6,Charles Adrian K.7,Tare Marianne89,Arnolda Leonard F.10,Ganss Ruth1

Affiliation:

1. Harry Perkins Institute of Medical Research, Centre for Medical Research, The University of Western Australia, Nedlands, Western Australia 6009, Australia.

2. Royal Perth Hospital, Perth, Western Australia 6009, Australia.

3. School of Women’s and Children’s Health, Faculty of Medicine, University of New South Wales, Sydney, New South Wales 2031, Australia.

4. Physiology, School of Medical Sciences, University of New South Wales, Sydney, New South Wales 2052, Australia.

5. Faculty of Science, Health, Education and Engineering, University of the Sunshine Coast, Maroochydore, Queensland 4558, Australia.

6. School of Medicine and Pharmacology, The University of Western Australia & Cardiology Department, Royal Perth Hospital, Perth, Western Australia 6000, Australia.

7. Princess Margaret Hospital and School of Paediatrics and Child Health, The University of Western Australia, Perth, Western Australia 6008, Australia.

8. Department of Physiology, Monash University, Clayton, Victoria 3800, Australia.

9. School of Rural Health, Monash University, Churchill, Victoria 3842, Australia.

10. Medical School, Australian National University, and Cardiology Department, Canberra Hospital, Canberra, Australian Capital Territory 2606, Australia.

Abstract

Reduction of vascular RGS5 causes hypertension and preeclampsia, which can be reversed by PPAR agonist treatment in mouse models.

Funder

National Health and Medical Research Council of Australia

Royal Perth Hospital Medical Research Foundation

Harry Perkins Institute of Medical Research

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号  京ICP备18003416号-3