Increased in Vivo Amyloid-β42 Production, Exchange, and Loss in Presenilin Mutation Carriers-Reference-Cited by-同舟云学术

Increased in Vivo Amyloid-β42 Production, Exchange, and Loss in Presenilin Mutation Carriers

Published:2013-06-12 Issue:189 Volume:5 Page:
ISSN:1946-6234
Container-title:Science Translational Medicine
language:en
Short-container-title:Sci. Transl. Med.

Author:

Potter Rachel¹,Patterson Bruce W.²,Elbert Donald L.³,Ovod Vitaliy¹,Kasten Tom¹,Sigurdson Wendy¹⁴,Mawuenyega Kwasi¹,Blazey Tyler⁴⁵,Goate Alison⁴⁶⁷,Chott Robert²,Yarasheski Kevin E.²,Holtzman David M.¹⁴⁶,Morris John C.¹⁴⁶,Benzinger Tammie L. S.⁴⁵⁸,Bateman Randall J.¹⁴⁶

Affiliation:

1. Department of Neurology, Washington University School of Medicine, 660 South Euclid Avenue, St. Louis, MO 63110, USA.

2. Department of Medicine, Washington University School of Medicine, St. Louis, MO 63110, USA.

3. Department of Biomedical Engineering, Washington University in St. Louis, One Brookings Drive, St. Louis, MO 63130, USA.

4. Knight Alzheimer’s Disease Research Center, Washington University School of Medicine, St. Louis, MO 63110, USA.

5. Department of Radiology, Washington University School of Medicine, St. Louis, MO 63110, USA.

6. Hope Center for Neurological Disorders, Washington University School of Medicine, St. Louis, MO 63110, USA.

7. Department of Psychiatry, Washington University School of Medicine, St. Louis, MO 63110, USA.

8. Department of Neurological Surgery, Washington University School of Medicine, St. Louis, MO 63110, USA.

Abstract

Aβ42 kinetics are altered in the central nervous system of patients with autosomal dominant Alzheimer’s disease.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine

Reference44 articles.

1. Autosomal-dominant Alzheimer's disease: a review and proposal for the prevention of Alzheimer's disease

2. Clinical and Biomarker Changes in Dominantly Inherited Alzheimer's Disease

3. The Amyloid Hypothesis of Alzheimer's Disease: Progress and Problems on the Road to Therapeutics

4. The Presenilins in Alzheimer's Disease--Proteolysis Holds the Key

5. The Profile of Soluble Amyloid β Protein in Cultured Cell Media

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