The Role of Nogo and the Mitochondria–Endoplasmic Reticulum Unit in Pulmonary Hypertension-Reference-Cited by-同舟云学术

The Role of Nogo and the Mitochondria–Endoplasmic Reticulum Unit in Pulmonary Hypertension

Published:2011-06-22 Issue:88 Volume:3 Page:
ISSN:1946-6234
Container-title:Science Translational Medicine
language:en
Short-container-title:Sci. Transl. Med.

Author:

Sutendra Gopinath¹,Dromparis Peter¹,Wright Paulette²,Bonnet Sébastien¹³,Haromy Alois¹,Hao Zhengrong²,McMurtry M. Sean¹,Michalak Marek⁴,Vance Jean E.¹,Sessa William C.²,Michelakis Evangelos D.¹

Affiliation:

1. Department of Medicine, University of Alberta, Edmonton, Alberta T6G 2B7, Canada.

2. Department of Pharmacology, Yale University School of Medicine, New Haven, CT 06520, USA.

3. Department of Medicine, Laval University, Quebec, Quebec G1R 2J6, Canada.

4. Department of Biochemistry, University of Alberta, Edmonton, Alberta T6G 2R7, Canada.

Abstract

The endoplasmic reticulum stress protein Nogo is a culprit in the mitochondrial defects that characterize pulmonary hypertension, pointing to therapeutic drug targets.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine

Reference51 articles.

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5. The role of mitochondria in pulmonary vascular remodeling

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