Fibronectin EDA Promotes Chronic Cutaneous Fibrosis Through Toll-Like Receptor Signaling-Reference-Cited by-同舟云学术

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Fibronectin EDA Promotes Chronic Cutaneous Fibrosis Through Toll-Like Receptor Signaling

Published:2014-04-16 Issue:232 Volume:6 Page:
ISSN:1946-6234
Container-title:Science Translational Medicine
language:en
Short-container-title:Sci. Transl. Med.

Author:

Bhattacharyya Swati¹,Tamaki Zenshiro¹,Wang Wenxia¹,Hinchcliff Monique¹,Hoover Paul²,Getsios Spiro²,White Eric S.³,Varga John¹

Affiliation:

1. Division of Rheumatology, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA.

2. Department of Dermatology, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA.

3. Department of Internal Medicine, University of Michigan, Ann Arbor, MI 48109–5642, USA.

Abstract

Fibronectin EDA is an endogenous TLR4 ligand in scleroderma.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine

Reference71 articles.

1. Targeted therapies for systemic sclerosis

2. Narrative Review: Fibrotic Diseases: Cellular and Molecular Mechanisms and Novel Therapies

3. Transforming growth factor β (TGFβ) causes a persistent increase in steady-state amounts of type I and type III collagen and fibronectin mRNAs in normal human dermal fibroblasts

4. Systemic sclerosis: a prototypic multisystem fibrotic disorder

5. Recent Developments in Myofibroblast Biology

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