The proinflammatory role of HECTD2 in innate immunity and experimental lung injury-Reference-Cited by-同舟云学术

The proinflammatory role of HECTD2 in innate immunity and experimental lung injury

Published:2015-07-08 Issue:295 Volume:7 Page:
ISSN:1946-6234
Container-title:Science Translational Medicine
language:en
Short-container-title:Sci. Transl. Med.

Author:

Coon Tiffany A.¹,McKelvey Alison C.¹,Lear Travis¹,Rajbhandari Shristi¹,Dunn Sarah R.¹,Connelly William¹,Zhao Joe Y.¹,Han SeungHye¹,Liu Yuan¹,Weathington Nathaniel M.¹,McVerry Bryan J.¹,Zhang Yingze¹,Chen Bill B.¹²

Affiliation:

1. Department of Medicine, Acute Lung Injury Center of Excellence, University of Pittsburgh, Pittsburgh, PA 15213, USA.

2. Vascular Medicine Institute, University of Pittsburgh, Pittsburgh, PA 15213, USA.

Abstract

Antagonizing the E3 ligase HECTD2 reduces inflammation severity and experimental lung injury.

Funder

NIH

University of Pittsburgh Vascular Medicine Institute

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine

Reference41 articles.

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3. Proinflammatory Stimuli Induce IKKα-Mediated Phosphorylation of PIAS1 to Restrict Inflammation and Immunity

4. Inhibition of Stat1-mediated gene activation by PIAS1

5. Protein Inhibitor of Activated STAT 1 (PIAS1) Is Identified as the SUMO E3 Ligase of CCAAT/Enhancer-Binding Protein β (C/EBPβ) during Adipogenesis

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