SETDB1-dependent heterochromatin stimulates alternative lengthening of telomeres-Reference-Cited by-同舟云学术

SETDB1-dependent heterochromatin stimulates alternative lengthening of telomeres

Published:2019-05-03 Issue:5 Volume:5 Page:
ISSN:2375-2548
Container-title:Science Advances
language:en
Short-container-title:Sci. Adv.

Author:

Gauchier Mathilde¹^ORCID,Kan Sophie¹^ORCID,Barral Amandine¹,Sauzet Sandrine¹,Agirre Eneritz¹^ORCID,Bonnell Erin²,Saksouk Nehmé¹^ORCID,Barth Teresa K.³,Ide Satoru¹,Urbach Serge⁴^ORCID,Wellinger Raymund J.²^ORCID,Luco Reini F.¹^ORCID,Imhof Axel³^ORCID,Déjardin Jérôme¹^ORCID

Affiliation:

1. Institute of Human Genetics CNRS-Université de Montpellier UMR 9002, 141 rue de la Cardonille, Montpellier 34000, France.

2. Department of Microbiology and Infectious Diseases, PRAC-Université de Sherbrooke 3201 Jean-Mignault, Sherbrooke, Qc J1E 4K8, Canada.

3. Munich Centre of Integrated Protein Science and Division of Molecular Biology Biomedical Center, Faculty of Medicine, LMU Munich, Großhaderner Str.9 82152 Planegg, Martinsried, Germany.

4. Functional Proteomics Facility, Institute of Functional Genomics, 141 rue de la Cardonille, 34000 Montpellier, France.

Abstract

Atypical heterochromatin forms on telomeres and correlates with ALT activities.

Funder

Merck

Association pour la Recherche sur le Cancer

European Research Council

Deutsche Forschungsgemeinschaft

Institut National Du Cancer

FOndation Schlumberger pour l’Education et la Recherche

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

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