Malaria parasite infection compromises colonization resistance to an enteric pathogen by reducing gastric acidity

Author:

Walker Gregory T.1ORCID,Yang Guiyan12,Tsai Julia Y.13,Rodriguez Jorge L.1ORCID,English Bevin C.1ORCID,Faber Franziska14ORCID,Souvannaseng Lattha156,Butler Brian P.6ORCID,Tsolis Renée M.1ORCID

Affiliation:

1. Department of Medical Microbiology and Immunology, School of Medicine, University of California Davis, Davis, CA, USA.

2. College of Veterinary Medicine, China Agricultural University, Beijing 100193, China.

3. School of Veterinary Medicine, University of California Davis, Davis, CA, USA.

4. Institute for Molecular Infection Biology (IMIB), Faculty of Medicine, University of Würzburg, D-97080 Würzburg, Germany.

5. Mouse Biology Program, University of California Davis, Davis, CA, USA.

6. Department of Pathobiology, School of Veterinary Medicine, St. George’s University, Grenada, West Indies.

Abstract

In mice, the host response to malaria weakens colonization resistance to an enteric pathogen by suppressing gastric acidity.

Funder

National Institutes of Health

National Institute of Allergy and Infectious Diseases

China Scholarship Council

St George’s University Postdoctoral Scholars Program

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

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