The road to death: Caspases, cleavage, and pores
Author:
Affiliation:
1. Physiological Chemistry Department, Genentech, 1 DNA Way, South San Francisco, CA 94080, USA.
Abstract
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Link
https://www.science.org/doi/pdf/10.1126/sciadv.adi2011
Reference12 articles.
1. Fas- and Tumor Necrosis Factor-induced Apoptosis Is Inhibited by the Poxvirus crmA Gene Product
2. Yama/CPP32β, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase
3. FADD, a novel death domain-containing protein, interacts with the death domain of fas and initiates apoptosis
4. FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling Complex
5. Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell Death
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