Mammalian EAK-7 activates alternative mTOR signaling to regulate cell proliferation and migration-Reference-Cited by-同舟云学术

Mammalian EAK-7 activates alternative mTOR signaling to regulate cell proliferation and migration

Published:2018-05-04 Issue:5 Volume:4 Page:
ISSN:2375-2548
Container-title:Science Advances
language:en
Short-container-title:Sci. Adv.

Author:

Nguyen Joe Truong¹²^ORCID,Ray Connor¹²^ORCID,Fox Alexandra Lucienne¹²^ORCID,Mendonça Daniela Baccelli¹²^ORCID,Kim Jin Koo¹²³^ORCID,Krebsbach Paul H.¹²³^ORCID

Affiliation:

1. Department of Biologic and Materials Sciences, University of Michigan, Ann Arbor, MI 48105, USA.

2. Biointerfaces Institute, University of Michigan, Ann Arbor, MI 48105, USA.

3. Section of Periodontics, University of California, Los Angeles, Los Angeles, CA 90095, USA.

Abstract

Mammalian EAK-7 forms an alternative mTOR complex.

Funder

National Institute of Dental and Craniofacial Research

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference43 articles.

1. EAK-7 controls development and life span by regulating nuclear DAF-16/FoxO activity;Alam H.;Cell Metab.,2010

2. Making new contacts: the mTOR network in metabolism and signalling crosstalk

3. mTOR Signaling in Growth, Metabolism, and Disease

4. Rapamycin-FKBP specifically blocks growth-dependent activation of and signaling by the 70 kd S6 protein kinases

5. Targets for Cell Cycle Arrest by the Immunosuppressant Rapamycin in Yeast

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