Noncanonical function of an autophagy protein prevents spontaneous Alzheimer’s disease-Reference-Cited by-同舟云学术

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Noncanonical function of an autophagy protein prevents spontaneous Alzheimer’s disease

Published:2020-08-14 Issue:33 Volume:6 Page:
ISSN:2375-2548
Container-title:Science Advances
language:en
Short-container-title:Sci. Adv.

Author:

Heckmann Bradlee L.¹^ORCID,Teubner Brett J. W.²,Boada-Romero Emilio¹^ORCID,Tummers Bart¹,Guy Clifford¹^ORCID,Fitzgerald Patrick¹,Mayer Ulrike³^ORCID,Carding Simon⁴,Zakharenko Stanislav S.²^ORCID,Wileman Thomas⁴⁵^ORCID,Green Douglas R.¹^ORCID

Affiliation:

1. Department of Immunology, St. Jude Children’s Research Hospital, Memphis, TN, USA.

2. Department of Developmental Neurobiology, St. Jude Children’s Research Hospital, Memphis, TN, USA.

3. School of Biological Sciences, University of East Anglia, Norwich, Norfolk, UK.

4. Quadram Institute of Bioscience, Norwich, Norfolk, UK.

5. Norwich Medical School, University of East Anglia, Norwich, Norfolk, UK.

Abstract

Mice with mutant ATG16L develop spontaneous, severe Alzheimer’s-like disease with age, despite normal autophagy.

Funder

National Institutes of Health

National Institute of Mental Health

National Cancer Institute

National Institute of Allergy and Infectious Diseases

Biotechnology and Biological Sciences Research Council

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference62 articles.

1. Alzheimer Disease: An Update on Pathobiology and Treatment Strategies

2. Autophagy and Neurodegeneration: Pathogenic Mechanisms and Therapeutic Opportunities

3. Genome-wide analysis reveals mechanisms modulating autophagy in normal brain aging and in Alzheimer's disease

4. Autophagy and Aging

5. Microglial Beclin 1 Regulates Retromer Trafficking and Phagocytosis and Is Impaired in Alzheimer’s Disease

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