Shared transcriptional profiles of atypical B cells suggest common drivers of expansion and function in malaria, HIV, and autoimmunity

Author:

Holla Prasida1ORCID,Dizon Brian2ORCID,Ambegaonkar Abhijit A.1ORCID,Rogel Noga3,Goldschmidt Ella3,Boddapati Arun K.4,Sohn Haewon1,Sturdevant Dan5,Austin James W.6,Kardava Lela6ORCID,Yuesheng Li7ORCID,Liu Poching7,Moir Susan6ORCID,Pierce Susan K.1ORCID,Madi Asaf3ORCID

Affiliation:

1. Laboratory of Immunogenetics, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Rockville, MD, USA.

2. National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, MD, USA.

3. Department of Pathology, Sackler School of Medicine, Tel Aviv University, Israel.

4. NIAID Collaborative Bioinformatics Resource, National Institutes of Health, Bethesda, MD, USA.

5. RML Genomics Unit, Research Technologies Section, Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, MT, USA.

6. Laboratory of Immunoregulation, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, USA.

7. National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, MD, USA.

Abstract

Atypical B cells represent an IFN-γ–driven B cell lineage that may be similarly expanded in malaria, HIV, and autoimmunity.

Funder

Division of Intramural Research, National Institute of Allergy and Infectious Diseases

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号  京ICP备18003416号-3