Blood-stage malaria parasites manipulate host innate immune responses through the induction of sFGL2-Reference-Cited by-同舟云学术

Blood-stage malaria parasites manipulate host innate immune responses through the induction of sFGL2

Published:2020-02-28 Issue:9 Volume:6 Page:
ISSN:2375-2548
Container-title:Science Advances
language:en
Short-container-title:Sci. Adv.

Author:

Fu Yong¹^ORCID,Ding Yan¹,Wang Qinghui²,Zhu Feng¹,Tan Yulong³^ORCID,Lu Xiao¹,Guo Bo¹,Zhang Qingfeng⁴,Cao Yaming²,Liu Taiping¹^ORCID,Cui Liwang⁵^ORCID,Xu Wenyue¹^ORCID

Affiliation:

1. Department of Pathogenic Biology, Army Medical University (The Third Military Medical University), Chongqing 400038, P.R. China.

2. Department of Immunology, College of Basic Medical Sciences, China Medical University, Shenyang, Liaoning, P.R. China.

3. Department of Tropical Medicine, Army Medical University (The Third Military Medical University), Chongqing 400038, P.R. China.

4. Research Center for Translational Medicine, Key Laboratory of Arrhythmias of the Ministry of Education of China, East Hospital, Tongji University School of Medicine, Shanghai, P.R. China.

5. Department of Internal Medicine, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA.

Abstract

Malaria parasite uses sFGL2 secreted mainly by T regs to subvert the host innate immune response for its survival.

Funder

National Natural Science Foundation of China

National Key Research and Development Program of China Stem Cell and Translational Research

Southeast Asia Malaria Research Center

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference43 articles.

1. Induction of Proinflammatory Responses in Macrophages by the Glycosylphosphatidylinositols of Plasmodium falciparum

2. Toll-like receptor 9 mediates innate immune activation by the malaria pigment hemozoin

3. Pure Hemozoin Is Inflammatory In Vivo and Activates the NALP3 Inflammasome via Release of Uric Acid

4. Gamma-Globulin and Acquired Immunity to Human Malaria

5. CD4+ T cells and B cells are necessary for the transfer of protective immunity toPlasmodium chabaudi chabaudi

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