Suppression of diabetes by accumulation of non–islet-specific CD8 + effector T cells in pancreatic islets
Author:
Affiliation:
1. Type 1 Diabetes Center, La Jolla Institute for Allergy and Immunology, La Jolla, CA 92037, USA.
2. Wroclaw Research Centre EIT+, Wroclaw, Poland.
3. Novo Nordisk Diabetes Research and Development Center, Seattle, WA 98109, USA.
Abstract
Funder
National Institutes of Health
Publisher
American Association for the Advancement of Science (AAAS)
Subject
General Medicine,Immunology
Reference48 articles.
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3. Demonstration of islet-autoreactive CD8 T cells in insulitic lesions from recent onset and long-term type 1 diabetes patients
4. Molecular Anatomy and Number of Antigen Specific CD8 T Cells Required to Cause Type 1 Diabetes
5. Induction of Bystander T Cell Proliferation by Viruses and Type I Interferon in Vivo
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