IL-22 controls iron-dependent nutritional immunity against systemic bacterial infections-Reference-Cited by-同舟云学术

IL-22 controls iron-dependent nutritional immunity against systemic bacterial infections

Published:2017-02-24 Issue:8 Volume:2 Page:
ISSN:2470-9468
Container-title:Science Immunology
language:en
Short-container-title:Sci. Immunol.

Author:

Sakamoto Kei¹,Kim Yun-Gi¹,Hara Hideki¹,Kamada Nobuhiko²,Caballero-Flores Gustavo¹,Tolosano Emanuela³,Soares Miguel P.⁴,Puente José L.⁵,Inohara Naohiro¹,Núñez Gabriel¹

Affiliation:

1. Department of Pathology and Comprehensive Cancer Center, University of Michigan Medical School, Ann Arbor, MI 48109, USA.

2. Division of Gastroenterology, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, MI 48109, USA.

3. Molecular Biotechnology Center, Department of Molecular Biotechnology and Health Sciences, University of Torino, 10126 Torino, Italy.

4. Instituto Gulbenkian de Ciência, 2780-156 Oeiras, Portugal.

5. Departamento de Microbiología Molecular, Instituto de Biotecnología, Universidad Nacional Autónoma de México, Cuernavaca, Morelos 62210, México.

Abstract

IL-22 induces the heme scavenger hemopexin to limit iron availability to pathogens during infection. See the related Focus by Datta.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine,Immunology

Reference39 articles.

1. Iron in Infection and Immunity

2. Iron homeostasis in host defence and inflammation

3. Lipocalin 2-deficient mice exhibit increased sensitivity to Escherichia coli infection but not to ischemia-reperfusion injury

4. The physiology of lactoferrin

5. Hepcidin and the Iron-Infection Axis

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