Type III interferon is a critical regulator of innate antifungal immunity

Author:

Espinosa Vanessa1ORCID,Dutta Orchi12ORCID,McElrath Constance123ORCID,Du Peicheng45ORCID,Chang Yun-Juan45ORCID,Cicciarelli Bryan3ORCID,Pitler Amy3ORCID,Whitehead Ian3ORCID,Obar Joshua J.6ORCID,Durbin Joan E.17ORCID,Kotenko Sergei V.13,Rivera Amariliz18ORCID

Affiliation:

1. Center for Immunity and Inflammation, New Jersey Medical School, Rutgers Biomedical and Health Sciences (RBHS), Newark, NJ 07103, USA.

2. Graduate School of Biomedical Sciences, RBHS, Newark, NJ 07103, USA.

3. Department of Microbiology, Biochemistry and Molecular Genetics, RBHS, Newark, NJ 07103, USA.

4. Genomics Research Program, RBHS, Newark, NJ 07103, USA.

5. High Performance and Research Computing, Office of Information Technology, RBHS, Newark, NJ 07103, USA.

6. Department of Microbiology and Immunology, Geisel School of Medicine at Dartmouth, Lebanon, NH 03756, USA.

7. Department of Pathology, New Jersey Medical School, RBHS, Newark, NJ 07103, USA.

8. Department of Pediatrics, New Jersey Medical School, RBHS, Newark, NJ 07103, USA.

Abstract

Type III interferons activate neutrophils to promote antifungal immune responses to Aspergillus fumigatus .

Funder

National Institute of Allergy and Infectious Diseases

Burroughs Wellcome Fund

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine,Immunology

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