TGF-β Induces Acetylation of Chromatin and of Ets-1 to Alleviate Repression of miR-192 in Diabetic Nephropathy-Reference-Cited by-同舟云学术

TGF-β Induces Acetylation of Chromatin and of Ets-1 to Alleviate Repression of miR-192 in Diabetic Nephropathy

Published:2013-06-04 Issue:278 Volume:6 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Kato Mitsuo¹,Dang Varun¹,Wang Mei¹,Park Jung Tak¹,Deshpande Supriya¹²,Kadam Swati²,Mardiros Armen²,Zhan Yumei³,Oettgen Peter³,Putta Sumanth¹,Yuan Hang¹,Lanting Linda¹,Natarajan Rama¹²

Affiliation:

1. Department of Diabetes and Division of Molecular Diabetes Research, Beckman Research Institute of the City of Hope, Duarte, CA 91010, USA.

2. Irell & Manella Graduate School of Biological Sciences, Beckman Research Institute of the City of Hope, Duarte, CA 91010, USA.

3. Division of Cardiology, and Molecular and Vascular Medicine, Center for Vascular Biology Research, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA 02115, USA.

Abstract

Acetylation of chromatin and Ets-1 contributes to induction of miR-192 , a microRNA involved in kidney pathogenesis.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference59 articles.

1. MicroRNAs as mediators and therapeutic targets in chronic kidney disease

2. MicroRNAs and the glomerulus

3. MicroRNA-192 in diabetic kidney glomeruli and its function in TGF-beta-induced collagen expression via inhibition of E-box repressors

4. Inhibiting MicroRNA-192 Ameliorates Renal Fibrosis in Diabetic Nephropathy

5. MicroRNA-29c Is a Signature MicroRNA under High Glucose Conditions That Targets Sprouty Homolog 1, and Its in Vivo Knockdown Prevents Progression of Diabetic Nephropathy

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