TGF-β promotes PI3K-AKT signaling and prostate cancer cell migration through the TRAF6-mediated ubiquitylation of p85α-Reference-Cited by-同舟云学术

TGF-β promotes PI3K-AKT signaling and prostate cancer cell migration through the TRAF6-mediated ubiquitylation of p85α

Published:2017-07-04 Issue:486 Volume:10 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Hamidi Anahita¹^ORCID,Song Jie²^ORCID,Thakur Noopur¹,Itoh Susumu³^ORCID,Marcusson Anders¹,Bergh Anders²^ORCID,Heldin Carl-Henrik¹^ORCID,Landström Maréne¹²^ORCID

Affiliation:

1. Ludwig Institute for Cancer Research and Science for Life Laboratory, Uppsala University, Uppsala SE 751 24, Sweden.

2. Unit of Pathology, Department of Medical Biosciences, Umeå University, Umeå SE 901 85, Sweden.

3. Laboratory of Biochemistry, Showa Pharmaceutical University, Tokyo 194-8543, Japan.

Abstract

The TGF-β signaling pathways are not as insulated from each other as was once thought.

Funder

Knut och Alice Wallenbergs Stiftelse

Swedish Research Council for Health, Working Life and Welfare

Swedish Cancer Society

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference59 articles.

1. TGFβ in Cancer

2. Mechanism of TGF-β signaling to growth arrest, apoptosis, and epithelial–mesenchymal transition

3. TGF-β signalling from cell membrane to nucleus through SMAD proteins

4. Non-Smad signaling pathways

5. Non-Smad pathways in TGF-β signaling

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