The TNF Family Member 4-1BBL Sustains Inflammation by Interacting with TLR Signaling Components During Late-Phase Activation-Reference-Cited by-同舟云学术

The TNF Family Member 4-1BBL Sustains Inflammation by Interacting with TLR Signaling Components During Late-Phase Activation

Published:2013-10 Issue:295 Volume:6 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Ma Jianhui¹,Bang Bo-Ram¹,Lu Jiawei¹²,Eun So-Young³,Otsuka Motoyuki⁴,Croft Michael³,Tobias Peter¹,Han Jiahuai²,Takeuchi Osamu⁵,Akira Shizuo⁶,Karin Michael⁷,Yagita Hideo⁸,Kang Young Jun¹

Affiliation:

1. Department of Immunology and Microbial Science, The Scripps Research Institute, La Jolla, CA 92037, USA.

2. State Key Laboratory of Cellular Stress Biology and School of Life Sciences, Xiamen University, Xiamen, Fujian 361005, China.

3. Division of Immune Regulation, La Jolla Institute for Allergy and Immunology, La Jolla, CA 92037, USA.

4. Department of Gastroenterology, Graduate School of Medicine, University of Tokyo, Tokyo 113-8655, Japan.

5. Laboratory of Infection and Prevention, Institute for Virus Research, Kyoto University, Kyoto 606-8507, Japan.

6. Laboratory of Host Defense, World Premier International Immunology Frontier Research Center, Osaka University, Osaka 565-0871, Japan.

7. Laboratory of Gene Regulation and Signal Transduction, Department of Pharmacology, School of Medicine, University of California, San Diego, La Jolla, CA 92093, USA.

8. Department of Immunology, Juntendo University School of Medicine, Tokyo 113-8421, Japan.

Abstract

Limiting the late phase of the macrophage response to microbial products can prevent lethal sepsis in mice.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

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