PI3K and Chemotaxis: A Priming Issue?-Reference-Cited by-同舟云学术

PI3K and Chemotaxis: A Priming Issue?

Published:2011-04-26 Issue:170 Volume:4 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Afonso Philippe V.¹,Parent Carole A.¹

Affiliation:

1. Laboratory of Cellular and Molecular Biology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.

Abstract

The differentiation or “priming” status of cells may explain contradictory findings for the role of phosphoinositide 3-kinases in chemotaxis.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference29 articles.

1. Mechanical modes of ‘amoeboid’ cell migration

2. Pleckstrin homology (PH) domains and phosphoinositides

3. Spatial and Temporal Regulation of 3-Phosphoinositides by PI 3-Kinase and PTEN Mediates Chemotaxis

4. G Protein Signaling Events Are Activated at the Leading Edge of Chemotactic Cells

5. Polarization of Chemoattractant Receptor Signaling During Neutrophil Chemotaxis

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2. A RAB35-p85/PI3K axis controls oscillatory apical protrusions required for efficient chemotactic migration;Nature Communications;2018-04-16

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4. Neutrophil migration in infection and wound repair: going forward in reverse;Nature Reviews Immunology;2016-05-27

5. Phosphatidylinositol-3,4-Bisphosphate and Its Binding Protein Lamellipodin Regulate Chemotaxis of Malignant B Lymphocytes;The Journal of Immunology;2015-12-22