TGF-β–mediated enhancement of T H 17 cell generation is inhibited by bone morphogenetic protein receptor 1α signaling-Reference-Cited by-同舟云学术

TGF-β–mediated enhancement of T H 17 cell generation is inhibited by bone morphogenetic protein receptor 1α signaling

Published:2018-08-28 Issue:545 Volume:11 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Browning Lauren M.¹^ORCID,Pietrzak Maciej²^ORCID,Kuczma Michal³,Simms Colin P.¹^ORCID,Kurczewska Agnieszka¹,Refugia Justin M.¹^ORCID,Lowery Dustin J.¹^ORCID,Rempala Grzegorz⁴^ORCID,Gutkin Dmitriy⁵,Ignatowicz Leszek³,Muranski Pawel⁶,Kraj Piotr¹^ORCID

Affiliation:

1. Department of Biological Sciences, Old Dominion University, Norfolk, VA 23529, USA.

2. Department of Biomedical Informatics, Ohio State University, Columbus, OH 43210, USA.

3. Institute for Biomedical Sciences, Georgia State University, Atlanta, GA 30303, USA.

4. College of Public Health, Ohio State University, Columbus, OH 43210, USA.

5. Department of Pathology, University of Pittsburgh, Pittsburgh, PA 15240, USA.

6. Columbia University Medical Center, New York, NY 10032, USA.

Abstract

BMP receptor 1α activation limits the TGF-β–mediated differentiation of CD4 + T cells that produce the cytokine IL-17.

Funder

NIH

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference95 articles.

1. TGFβ signalling in context

2. Bone Morphogenetic Proteins: A critical review

3. Bone morphogenetic proteins in inflammation, glucose homeostasis and adipose tissue energy metabolism

4. Immunoregulation by members of the TGFβ superfamily

5. TGF-β: A Master of All T Cell Trades

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