Dynamic pre-BCR homodimers fine-tune autonomous survival signals in B cell precursor acute lymphoblastic leukemia-Reference-Cited by-同舟云学术

Dynamic pre-BCR homodimers fine-tune autonomous survival signals in B cell precursor acute lymphoblastic leukemia

Published:2016-11-29 Issue:456 Volume:9 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Erasmus M. Frank¹²,Matlawska-Wasowska Ksenia²³,Kinjyo Ichiko¹²,Mahajan Avanika¹²,Winter Stuart S.²³,Xu Li⁴,Horowitz Michael⁴,Lidke Diane S.¹²,Wilson Bridget S.¹²

Affiliation:

1. Department of Pathology, University of New Mexico Health Sciences Center, Albuquerque, NM 87131, USA.

2. UNM Comprehensive Cancer Center, University of New Mexico Health Sciences Center, Albuquerque, NM 87131, USA.

3. Department of Pediatrics, University of New Mexico Health Sciences Center, Albuquerque, NM 87131, USA.

4. Sea Lane Biotechnologies, 2450 Bayshore Parkway, Mountain View, CA 94043, USA.

Abstract

Homotypic interactions between pre–B cell receptors generate survival signals in human acute lymphoblastic leukemia cells.

Funder

NIH

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference97 articles.

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2. Syk and pTyr'd: Signaling through the B cell antigen receptor

3. Role of the Syk autophosphorylation site and SH2 domains in B cell antigen receptor signaling.

4. Pre-B cell receptor–mediated activation of BCL6 induces pre-B cell quiescence through transcriptional repression of MYC

5. Ras orchestrates exit from the cell cycle and light-chain recombination during early B cell development

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