Bridging the Synaptic Cleft: Lessons from Orphan Glutamate Receptors-Reference-Cited by-同舟云学术

Bridging the Synaptic Cleft: Lessons from Orphan Glutamate Receptors

Published:2010-08-24 Issue:136 Volume:3 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Schmid Sabine M.¹,Hollmann Michael²

Affiliation:

1. Department of Cellular and Molecular Pharmacology, University of California, San Francisco, 600 16th Street, GH-N276, San Francisco, CA 94143, USA.

2. Department of Biochemistry I—Receptor Biochemistry, Ruhr-University Bochum, Universitätsstrasse 150, D-44780 Bochum, Germany.

Abstract

Three proteins act as a transsynaptic bridge at a major type of cerebellar synapse.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference35 articles.

1. Cell adhesion molecules: signalling functions at the synapse

2. Induction, assembly, maturation and maintenance of a postsynaptic apparatus

3. Cbln1 Is a Ligand for an Orphan Glutamate Receptor δ2, a Bidirectional Synapse Organizer

4. Trans-Synaptic Interaction of GluRδ2 and Neurexin through Cbln1 Mediates Synapse Formation in the Cerebellum

5. Impairment of motor coordination, Purkinje cell synapse formation, and cerebellar long-term depression in GluRδ2 mutant mice

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