The Specificity of Innate Immune Responses Is Enforced by Repression of Interferon Response Elements by NF-κB p50-Reference-Cited by-同舟云学术

The Specificity of Innate Immune Responses Is Enforced by Repression of Interferon Response Elements by NF-κB p50

Published:2011-02-22 Issue:161 Volume:4 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Cheng Christine S.¹²³,Feldman Kristyn E.¹²,Lee James¹²,Verma Shilpi⁴,Huang De-Bin²,Huynh Kim²,Chang Mikyoung⁵,Ponomarenko Julia V.⁶,Sun Shao-Cong⁵,Benedict Chris A.⁴,Ghosh Gourisankar²,Hoffmann Alexander¹²³

Affiliation:

1. Signaling Systems Laboratory, University of California–San Diego, 9500 Gilman Drive, La Jolla, CA 92093, USA.

2. Department of Chemistry and Biochemistry, University of California–San Diego, La Jolla, CA 92093, USA.

3. San Diego Center for Systems Biology, University of California–San Diego, La Jolla, CA 92093, USA.

4. Division of Immune Regulation, La Jolla Institute for Allergy and Immunology, La Jolla, CA 92037, USA.

5. Department of Immunology, University of Texas MD Anderson Cancer Center, 7455 Fannin Street, Houston, TX 77030, USA.

6. San Diego Supercomputer Center, University of California–San Diego, La Jolla, CA 92093, USA.

Abstract

Binding of NF-κB p50 homodimers to an interferon response element restricts interferon signaling and antiviral responses to only the appropriate stimuli.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference69 articles.

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4. Molecular Determinants of Crosstalk between Nuclear Receptors and Toll-like Receptors

5. Gene-specific control of inflammation by TLR-induced chromatin modifications

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