The death-inducing activity of RIPK1 is regulated by the pH environment-Reference-Cited by-同舟云学术

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The death-inducing activity of RIPK1 is regulated by the pH environment

Published:2020-05-12 Issue:631 Volume:13 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Moriwaki Kenta¹²^ORCID,Balaji Sakthi¹^ORCID,Ka-Ming Chan Francis¹³^ORCID

Affiliation:

1. Department of Pathology, Immunology and Microbiology Program, University of Massachusetts Medical School, Worcester, MA 01655, USA.

2. Department of Cell Biology, Osaka University Graduate School of Medicine, Suita, Osaka 565-0871, Japan.

3. Department of Immunology, Duke University School of Medicine, Durham, NC 27710, USA.

Abstract

Low extracellular pH suppresses RIPK1-dependent cell death.

Funder

National Institute of Allergy and Infectious Diseases

Takeda Science Foundation

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference58 articles.

1. Control of Life-or-Death Decisions by RIP1 Kinase

2. Programmed Necrosis in the Cross Talk of Cell Death and Inflammation

3. Induction of TNF Receptor I-Mediated Apoptosis via Two Sequential Signaling Complexes

4. Holding RIPK1 on the Ubiquitin Leash in TNFR1 Signaling

5. Checkpoints in TNF-Induced Cell Death: Implications in Inflammation and Cancer

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1. An acidic pH environment converts necroptosis to apoptosis;Biochemical and Biophysical Research Communications;2024-09

2. Necroptosis in immunity, tissue homeostasis, and cancer;Current Opinion in Immunology;2024-08

3. Deciphering the Molecular Nexus: An In-Depth Review of Mitochondrial Pathways and Their Role in Cell Death Crosstalk;Cells;2024-05-17

4. Gasdermin and MLKL necrotic cell death effectors: Signaling and diseases;Immunity;2024-03

5. Neutrophil metabolomics in severe COVID-19 reveal GAPDH as a suppressor of neutrophil extracellular trap formation;Nature Communications;2023-05-05

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