c-mip Impairs Podocyte Proximal Signaling and Induces Heavy Proteinuria-Reference-Cited by-同舟云学术

c-mip Impairs Podocyte Proximal Signaling and Induces Heavy Proteinuria

Published:2010-05-18 Issue:122 Volume:3 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Zhang Shao-yu¹²,Kamal Maud¹²,Dahan Karine¹²,Pawlak André¹²,Ory Virginie¹²,Desvaux Dominique¹²,Audard Vincent¹²,Candelier Marina¹²,BenMohamed Fatima¹²,Matignon Marie¹²,Christov Christo³⁴,Decrouy Xavier⁴,Bernard Veronique⁵,Mangiapan Gilles⁶,Lang Philippe¹²⁷⁸,Guellaën Georges¹²,Ronco Pierre⁹¹⁰¹¹,Sahali Djillali¹²⁷⁸

Affiliation:

1. INSERM, UMR 955, Equipe 21, F-94010 Créteil, France.

2. UMRS 955, Equipe 21, Université Paris-Est Créteil Val-de-Marne, F-94010 Créteil, France.

3. Assistance publique-Hôpitaux de Paris (AP-HP), Groupe hospitalier Henri Mondor-Albert Chenevier, Service d’histologie, Département de Pathologie, F-94010 Créteil, France.

4. INSERM, UMR 955, Plate-Forme d’Imagerie Cellulaire et Tissulaire, F-94010 Créteil, France.

5. Unité INSERM, UMR 952, CNRS UMR7224, Université Pierre et Marie Curie, F-75252 Paris, France.

6. Centre hospitalier intercommunal, Service de pneumologie, F-94010 Créteil, France.

7. AP-HP, Groupe hospitalier Henri Mondor–Albert Chenevier, Service de Néphrologie, F-94010 Créteil, France.

8. Institut francilien de recherche en néphrologie et transplantation Henri Mondor, F-94010 Créteil, France.

9. INSERM, UMRS-702, F-75020 Paris, France.

10. Université Pierre et Marie Curie–Paris 6, 75252 Paris Cedex 05, France.

11. AP-HP, Hôpital Tenon, Service de Néphrologie et Dialyses, F-75020 Paris, France.

Abstract

Overexpression of the protein c-mip in mice produces phenotypes similar to various idiopathic kidney disorders.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference43 articles.

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3. Caught flat-footed: podocyte damage and the molecular bases of focal glomerulosclerosis

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5. Positionally Cloned Gene for a Novel Glomerular Protein—Nephrin—Is Mutated in Congenital Nephrotic Syndrome

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