Brd4 Shields Chromatin from ATM Kinase Signaling Storms-Reference-Cited by-同舟云学术

Brd4 Shields Chromatin from ATM Kinase Signaling Storms

Published:2013-09-17 Issue:293 Volume:6 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Choi Serah¹²,Bakkenist Christopher J.²³

Affiliation:

1. UPMC Medical Education Transitional Year Residency Program, University of Pittsburgh School of Medicine, Hillman Cancer Center, Research Pavilion, Pittsburgh, PA 15213, USA.

2. Department of Radiation Oncology, University of Pittsburgh School of Medicine, Hillman Cancer Center, Research Pavilion, Pittsburgh, PA 15213, USA.

3. Department of Pharmacology and Chemical Biology, University of Pittsburgh School of Medicine, Hillman Cancer Center, Research Pavilion, Pittsburgh, PA 15213, USA.

Abstract

New studies identify chromatin-binding proteins that modulate ATM signaling in response to DNA damage.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference12 articles.

1. DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation

2. ATM and ATR Substrate Analysis Reveals Extensive Protein Networks Responsive to DNA Damage

3. Quantitative Phosphoproteomics Reveals Widespread Full Phosphorylation Site Occupancy During Mitosis

4. KAT5 tyrosine phosphorylation couples chromatin sensing to ATM signalling

5. The bromodomain protein Brd4 insulates chromatin from DNA damage signalling

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3. Phosphoproteomics Reveals AMPK Substrate Network in Response to DNA Damage and Histone Acetylation;Genomics, Proteomics & Bioinformatics;2021-02

4. Phosphoproteomics Reveals AMPK Substrate Network in Response to DNA Damage and Histone Acetylation;2020-07-02

5. ATM Is Required for the Repair of Oxaliplatin-Induced DNA Damage in Colorectal Cancer;Clinical Colorectal Cancer;2018-12