ABL kinases promote breast cancer osteolytic metastasis by modulating tumor-bone interactions through TAZ and STAT5 signaling-Reference-Cited by-同舟云学术

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ABL kinases promote breast cancer osteolytic metastasis by modulating tumor-bone interactions through TAZ and STAT5 signaling

Published:2016-02-02 Issue:413 Volume:9 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Wang Jun¹,Rouse Clay²,Jasper Jeff S.¹,Pendergast Ann Marie¹

Affiliation:

1. Department of Pharmacology and Cancer Biology, Duke University School of Medicine, Durham, NC 27710, USA.

2. Division of Laboratory Animal Resources, Duke University School of Medicine, Durham, NC 27710, USA.

Abstract

ABL kinase inhibitors could prevent breast cancer cells from breaking down bone to promote metastasis.

Funder

NIH

National Center for Advancing Translational Sciences

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference59 articles.

1. Regulation of cell migration and morphogenesis by Abl-family kinases: emerging mechanisms and physiological contexts

2. ABL Tyrosine Kinases: Evolution of Function, Regulation, and Specificity

3. The Abl family kinases: Mechanisms of regulation and signaling

4. The BCR-ABL Story: Bench to Bedside and Back

5. Role of ABL family kinases in cancer: from leukaemia to solid tumours

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