NF-κB activation in cardiac fibroblasts results in the recruitment of inflammatory Ly6C hi monocytes in pressure-overloaded hearts-Reference-Cited by-同舟云学术

NF-κB activation in cardiac fibroblasts results in the recruitment of inflammatory Ly6C hi monocytes in pressure-overloaded hearts

Published:2021-10-12 Issue:704 Volume:14 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Abe Hajime¹^ORCID,Tanada Yohei¹^ORCID,Omiya Shigemiki¹^ORCID,Podaru Mihai-Nicolae¹,Murakawa Tomokazu¹^ORCID,Ito Jumpei¹^ORCID,Shah Ajay M.¹^ORCID,Conway Simon J.²,Ono Masahiro³^ORCID,Otsu Kinya¹⁴^ORCID

Affiliation:

1. The School of Cardiovascular Medicine and Sciences, King’s College London British Heart Foundation Centre of Excellence, London SE5 9NU, UK.

2. Herman B. Wells Center for Pediatric Research, Department of Pediatrics, Indiana University School of Medicine, Indianapolis, IN 46202, USA.

3. Department of Life Sciences, Imperial College London, London SW7 2AZ, UK.

4. National Cerebral and Cardiovascular Center, 6-1 Kishibe-Shimmachi, Suita, Osaka 564-8565, Japan.

Abstract

A subset of cardiac fibroblasts responds to pressure overload by recruiting inflammatory monocytes to the heart.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference28 articles.

1. Global Public Health Burden of Heart Failure

2. Reappraising the role of inflammation in heart failure

3. Macrophage hypoxia signaling regulates cardiac fibrosis via Oncostatin M

4. The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions

5. Distinct roles of resident and nonresident macrophages in nonischemic cardiomyopathy

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