Global increase in replication fork speed during a p57 KIP2 -regulated erythroid cell fate switch-Reference-Cited by-同舟云学术

Global increase in replication fork speed during a p57 KIP2 -regulated erythroid cell fate switch

Published:2017-05-05 Issue:5 Volume:3 Page:
ISSN:2375-2548
Container-title:Science Advances
language:en
Short-container-title:Sci. Adv.

Author:

Hwang Yung¹,Futran Melinda¹,Hidalgo Daniel¹,Pop Ramona¹,Iyer Divya Ramalingam²^ORCID,Scully Ralph³^ORCID,Rhind Nicholas²^ORCID,Socolovsky Merav¹

Affiliation:

1. Department of Molecular, Cell and Cancer Biology, University of Massachusetts Medical School, Worcester, MA 01605, USA.

2. Department of Biochemistry and Molecular Pharmacology, University of Massachusetts Medical School, Worcester, MA 01605, USA.

3. Division of Hematology-Oncology, Department of Medicine, and Cancer Research Institute, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, MA 02115, USA.

Abstract

The switch from self-renewal to differentiation coincides with a shorter S phase in which replication forks are faster.

Funder

Leukemia and Lymphoma Society

National Institute of Diabetes and Digestive and Kidney Diseases

National Cancer Institute

National Institute of Health

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference82 articles.

1. Cdks, cyclins and CKIs: roles beyond cell cycle regulation

2. Interaction of myogenic factors and the retinoblastoma protein mediates muscle cell commitment and differentiation

3. Helper T Cell Differentiation Is Controlled by the Cell Cycle

4. Coordinating cell proliferation and differentiation

5. In search of “stemness”

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