Inhibition of Ras-Induced Proliferation and Cellular Transformation by p16 INK4-Reference-Cited by-同舟云学术

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Inhibition of Ras-Induced Proliferation and Cellular Transformation by p16 INK4

Published:1995-01-13 Issue:5195 Volume:267 Page:249-252
ISSN:0036-8075
Container-title:Science
language:en
Short-container-title:Science

Author:

Serrano Manuel¹,Gómez-Lahoz Enrique²,DePinho Ronald A.²,Beach David¹,Bar-Sagi Dafna³

Affiliation:

1. Howard Hughes Medical Institute, Cold Spring Harbor Laboratory, Cold Spring Harbor, NY 11724, USA.

2. Departments of Microbiology and Immunology and of Medicine, Albert Einstein College of Medicine, New York, NY 10461, USA.

3. Cold Spring Harbor Laboratory, Cold Spring Harbor, NY 1 1724, USA.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference44 articles.

1. ANDO, K, REGULATION OF G(1)/S TRANSITION BY CYCLIN-D2 AND CYCLIN-D3 IN HEMATOPOIETIC-CELLS, PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 90: 9571 (1993).

2. BALDIN, V, CYCLIN D1 IS A NUCLEAR-PROTEIN REQUIRED FOR CELL-CYCLE PROGRESSION IN G(1), GENES & DEVELOPMENT 7: 812 (1993).

3. BARTKOVA, J, CYCLIN D1 PROTEIN EXPRESSION AND FUNCTION IN HUMAN BREAST-CANCER, INTERNATIONAL JOURNAL OF CANCER 57: 353 (1994).

4. BATES, S, ABSENCE OF CYCLIN-D CDK COMPLEXES IN CELLS LACKING FUNCTIONAL RETINOBLASTOMA PROTEIN, ONCOGENE 9: 1633 (1994).

5. CHAMBERS, A.F., RAS-RESPONSIVE GENES AND TUMOR-METASTASIS, CRITICAL REVIEWS IN ONCOGENESIS 4: 95 (1993).

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