Genetic Determination of Phenotypic Variation in Sickle Cell Trait
Author:
Affiliation:
1. Department of Medical Genetics, Indiana University School of Medicine, Indianapolis 46202
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Reference31 articles.
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2. BOOKCHIN, R.M., CLINICAL FEATURES OF HEMOGLOBIN CHARLEM A NEW SICKLING HEMOGLOBIN VARIANT, ANNALS OF INTERNAL MEDICINE 68: 8 (1968).
3. BOYER, S, QUANTITATIVE DISPARITY BETWEEN HEMOGLOBINS - NATURAL MODELS FOR ANALYSIS OF CONTROL OF PROTEIN BIOSYNTHESIS, JOURNAL OF HEREDITY 58: 279 (1967).
4. DHERTE, P, HAEMOGLOBIN P IN A FAMILY IN THE BELGIAN CONGO, NATURE 184: 1133 (1959).
5. HILSE, K, GENE DUPLICATION AS BASIS FOR AMINO ACID AMBIGUITY IN ALPHA-CHAIN POLYPEPTIDES OF MOUSE HEMOGLOBINS, PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 61: 930 (1968).
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2. Complications Associated with Sickle Cell Trait: A Brief Narrative Review;The American Journal of Medicine;2009-06
3. Phenotypic Variation in Sickle Cell Trait;Scandinavian Journal of Haematology;2009-04-24
4. Effects of alpha-thalassemia and sickle polymerization tendency on the urine-concentrating defect of individuals with sickle cell trait.;Journal of Clinical Investigation;1991-12-01
5. Estimation of Genetical Parameters for a Quantitative Trait Subject to Major Gene Influences;Human Heredity;1987
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