Disruption of PDGF Receptor Trafficking by Mutation of Its PI-3 Linase Binding Sites-Reference-Cited by-同舟云学术

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Disruption of PDGF Receptor Trafficking by Mutation of Its PI-3 Linase Binding Sites

Published:1994-02-04 Issue:5147 Volume:263 Page:684-687
ISSN:0036-8075
Container-title:Science
language:en
Short-container-title:Science

Author:

Joly Marguerite¹,Kazlauskas Andrius²,Fay Fredric S.³,Corvera Silvia¹

Affiliation:

1. Program in Molecular Medicine and Department of Cell Biology, University of Massachusetts Medical School, Worcester, MA 01605.

2. National Jewish Hospital for Immunology and Respiratory Medicine, Denver, CO 80206.

3. Program in Molecular Medicine and Biomedical Imaging Group, University of Massachusetts Medical School, Worcester. MA 01655.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference33 articles.

1. PDGF-dependent tyrosine phosphorylation stimulates production of novel polyphosphoinositides in intact cells

2. BACKER, J.M., TYROSINE PHOSPHORYLATION OF THE INSULIN-RECEPTOR DURING INSULIN-STIMULATED INTERNALIZATION IN RAT HEPATOMA-CELLS, JOURNAL OF BIOLOGICAL CHEMISTRY 264: 1694 (1989).

3. CANTLEY, L.C., ONCOGENES AND SIGNAL TRANSDUCTION, CELL 64: 281 (1991).

4. Carrington, W. A., Non-Invasive Techniques in Cell Biology: 53 (1990).

5. Role of Phosphatidylinositol Kinase in PDGF Receptor Signal Transduction

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