Human Diabetes Associated with a Deletion of the Tyrosine Kinase Domain of the Insulin Receptor-Reference-Cited by-同舟云学术

Human Diabetes Associated with a Deletion of the Tyrosine Kinase Domain of the Insulin Receptor

Published:1989-07-07 Issue:4913 Volume:245 Page:63-66
ISSN:0036-8075
Container-title:Science
language:en
Short-container-title:Science

Author:

Taira Masato¹,Taira Masanori²,Hashimoto Naotake¹,Shimada Fumio¹,Suzuki Yoshifumi¹,Kanatsuka Azuma¹,Nakamura Fumiko³,Ebina Yousuke⁴,Tatibana Masamiti²,Makino Hideichi¹,Yoshida Sho¹

Affiliation:

1. Second Department of Internal Medicine, Chiba University School of Medicine, Inohana, Chiba 280, Japan.

2. Department of Biochemistry, Chiba University School of Medicine, Inohana, Chiba 280, Japan.

3. Department of Pediatrics, Shimoshizu National Hospital and Sanatorium, Yotsukaido, Chiba 284, Japan.

4. Department of Enzyme Genetics, Institute for Enzyme Research, University of Tokushima, Kuramotocho, Tokushima 770, Japan.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference24 articles.

1. Bell, G. I., Proceedings of the National Academy of Sciences of the United States of America 78: 5759 (1981).

2. CHOU, C.K., HUMAN INSULIN-RECEPTORS MUTATED AT THE ATP-BINDING SITE LACK PROTEIN TYROSINE KINASE-ACTIVITY AND FAIL TO MEDIATE POSTRECEPTOR EFFECTS OF INSULIN, JOURNAL OF BIOLOGICAL CHEMISTRY 262: 1842 (1987).

3. Base sequence studies of 300 nucleotide renatured repeated human DNA clones

4. EBINA, Y, THE HUMAN INSULIN-RECEPTOR CDNA - THE STRUCTURAL BASIS FOR HORMONE-ACTIVATED TRANSMEMBRANE SIGNALING, CELL 40: 747 (1985).

5. EBINA, Y, REPLACEMENT OF LYSINE RESIDUE 1030 IN THE PUTATIVE ATP-BINDING REGION OF THE INSULIN-RECEPTOR ABOLISHES INSULIN-STIMULATED AND ANTIBODY-STIMULATED GLUCOSE-UPTAKE AND RECEPTOR KINASE-ACTIVITY, PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 84: 704 (1987).

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