Absence of Pyruvate Decarboxylase Activity in Man: A Cause of Congenital Lactic Acidosis
Author:
Affiliation:
1. Departments of Medicine (Neurology) and Pediatrics, University of Washington School of Medicine, Seattle 98195
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Reference16 articles.
1. BAERLOCHER, K, INFANTILE LACTIC ACIDOSIS DUE TO HEREDITARY FRUCTOSE 1,6-DIPHOSPHATASE DEFICIENCY, HELVETICA PAEDIATRICA ACTA 26: 489 (1971).
2. BARRERA, C.R., ALPHA-KETO ACID DEHYDROGENASE COMPLEXES .16. STUDIES ON SUBUNIT STRUCTURE OF PYRUVATE DEHYDROGENASE COMPLEXES FROM BOVINE KIDNEY AND HEART, ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS 148: 343 (1972).
3. BLASS, J.P., CLINICAL STUDIES OF A PATIENT WITH PYRUVATE DECARBOXYLASE DEFICIENCY, ARCHIVES OF NEUROLOGY 25: 449 (1971).
4. BLASS, J.P., INHERITED DEFECT AFFECTING TRICARBOXYLIC ACID CYCLE IN A PATIENT WITH CONGENITAL LACTIC ACIDOSIS, JOURNAL OF CLINICAL INVESTIGATION 51: 1845 (1972).
5. BLASS, J.P., JOURNAL OF CLINICAL INVESTIGATION 49: 423 (1970).
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