Decreased Activity and Energy Balance in the Hereditary Obesity-Diabetes Syndrome of Mice
Author:
Affiliation:
1. Department of Nutrition, Harvard School of Public Health, Boston, Massachusetts
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Reference13 articles.
1. BLEISCH, V.R., FAMILIAL DIABETES MELLITUS IN MICE, ASSOCIATED WITH INSULIN RESISTANCE, OBESITY, AND HYPERPLASIA OF THE ISLANDS OF LANGERHANS, AMERICAN JOURNAL OF PATHOLOGY 28: 369 (1952).
2. GOLDBERG, R.C., NORMAL IODINE UPTAKE AND ANOXIA RESISTANCE ACCOMPANYING APPARENT HYPOMETABOLISM IN HEREDITARY OBESE-HYPERGLYCEMIC SYNDROME, PROCEEDINGS OF THE SOCIETY FOR EXPERIMENTAL BIOLOGY AND MEDICINE 81: 323 (1952).
3. GUGGENHEIM, K, STUDIES OF PYRUVATE AND ACETATE METABOLISM IN THE HEREDITARY OBESITY-DIABETES SYNDROME OF MICE, JOURNAL OF BIOLOGICAL CHEMISTRY 198: 259 (1952).
4. OBESE, A NEW MUTATION IN THE HOUSE MOUSE*
5. MAYER, J, BASAL OXYGEN CONSUMPTION OF HEREDITARILY OBESE AND DIABETIC MICE, ENDOCRINOLOGY 50: 318 (1952).
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