Defective Epithelial Chloride Transport in a Gene-Targeted Mouse Model of Cystic Fibrosis
Author:
Affiliation:
1. Division of Pulmonary Diseases, Department of Medicine, University of North Carolina, Chapel Hill, NC 27514
2. Department of Medicine, University of North Carolina, Chapel Hill, NC 27514
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Reference36 articles.
1. Demonstration That CFTR Is a Chloride Channel by Alteration of Its Anion Selectivity
2. BAXTER, P, ENHANCED INTESTINAL GLUCOSE AND ALANINE TRANSPORT IN CYSTIC-FIBROSIS, GUT 31: 817 (1990).
3. BAXTER, P.S., ABNORMAL JEJUNAL POTENTIAL DIFFERENCE IN CYSTIC-FIBROSIS, LANCET 1: 464 (1989).
4. BEAR, C.E., PURIFICATION AND FUNCTIONAL RECONSTITUTION OF THE CYSTIC-FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR (CFTR), CELL 68: 809 (1992).
5. BERSCHNEIDER, H.M., ALTERED INTESTINAL CHLORIDE TRANSPORT IN CYSTIC-FIBROSIS, FASEB JOURNAL 2: 2625 (1988).
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