AP1/ jun Function Is Differentially Induced in Promotion-Sensitive and Resistant JB6 Cells
Author:
Affiliation:
1. Johns Hopkins University, Department of Biology, Baltimore, MD 21218.
2. Cell Biology Section, Laboratory of Viral Carnoesis, NationalCancer Institute, Frederick, MD 21701.
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Reference38 articles.
1. ALWINE, J.C., TRANSIENT GENE-EXPRESSION CONTROL - EFFECTS OF TRANSFECTED DNA STABILITY AND TRANS-ACTIVATION BY VIRAL EARLY PROTEINS, MOLECULAR AND CELLULAR BIOLOGY 5: 1034 (1985).
2. ANGEL, P, PHORBOL ESTER INDUCIBLE GENES CONTAIN A COMMON CIS ELEMENT RECOGNIZED BY A TPA-MODULATED TRANS-ACTING FACTOR, CELL 49: 729 (1987).
3. The jun proto-oncogene is positively autoregulated by its product, Jun/AP-1
4. ANGEL, P, ONCOGENE JUN ENCODES A SEQUENCE-SPECIFIC TRANS-ACTIVATOR SIMILAR TO AP-1, NATURE 332: 166 (1988).
5. BERNSTEIN L.R. unpublished data.
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