Requirement for CD8 + Cells in T Cell Receptor Peptide-Induced Clonal Unresponsiveness
Author:
Affiliation:
1. Department of Medicine, Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, CA 94305.
2. ImmuLogic Pharmaceutical Corporation, Palo Alto, CA 94304.
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Reference21 articles.
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2. Cold Spring Harbor Symposia on Quantitative Biology 54: 879 (1989).
3. DESQUENNECLARK, L, T-CELL RECEPTOR PEPTIDE IMMUNIZATION LEADS TO ENHANCED AND CHRONIC EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS, PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 88: 7219 (1991).
4. DIALYNAS, D.P., CHARACTERIZATION OF THE MURINE T-CELL SURFACE-MOLECULE, DESIGNATED L3T4, IDENTIFIED BY MONOCLONAL-ANTIBODY GK1.5 - SIMILARITY OF L3T4 TO THE HUMAN LEU-3-T4 MOLECULE, JOURNAL OF IMMUNOLOGY 131: 2445 (1983).
5. HASHIM, G.A., ANTIBODIES SPECIFIC FOR VB8 RECEPTOR PEPTIDE SUPPRESS EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS, JOURNAL OF IMMUNOLOGY 144: 4621 (1990).
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