Neutrophil Mac-1 and MEL-14 Adhesion Proteins Inversely Regulated by Chemotactic Factors
Author:
Affiliation:
1. Department of Pathology, Stanford University, Stanford, CA 94305 and the Veterans Administration Medical Center, Palo Alto, CA 94304.
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Reference33 articles.
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2. ARFORS, K.E., A MONOCLONAL-ANTIBODY TO THE MEMBRANE GLYCOPROTEIN COMPLEX CD18 INHIBITS POLYMORPHONUCLEAR LEUKOCYTE ACCUMULATION AND PLASMA LEAKAGE INVIVO, BLOOD 69: 338 (1987).
3. BUCHANAN, M.R., STUDIES ON THE INTERACTION BETWEEN GP-180-DEFICIENT NEUTROPHILS AND VASCULAR ENDOTHELIUM, BLOOD 60: 160 (1982).
4. BUYON, J.P., DISSOCIATION BETWEEN INCREASED SURFACE EXPRESSION OF GP165/95 AND HOMOTYPIC NEUTROPHIL AGGREGATION, JOURNAL OF IMMUNOLOGY 140: 3156 (1988).
5. FAJARDO, L.F., HYPERTHERMIA INHIBITS ANGIOGENESIS, RADIATION RESEARCH 114: 297 (1988).
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