Rad: a Member of the Ras Family Overexpressed in Muscle of Type II Diabetic Humans
Author:
Affiliation:
1. Research Division, Joslin Diabetes Center, and Department of Medicine, Harvard Medical School, Boston, MA 02215.
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Reference59 articles.
1. ALBRIGHT, C.F., CHARACTERIZATION OF A GUANINE-NUCLEOTIDE DISSOCIATION STIMULATOR FOR A RAS-RELATED GTPASE, EMBO JOURNAL 12: 339 (1993).
2. BALDINI, G, CLONING OF A RAB3 ISOTYPE PREDOMINATELY EXPRESSED IN ADIPOCYTES, PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 89: 5049 (1992).
3. BALLINGER, S.W., MATERNALLY TRANSMITTED DIABETES AND DEAFNESS ASSOCIATED WITH A 10.4 KB MITOCHONDRIAL-DNA DELETION, NATURE GENETICS 1: 11 (1992).
4. Binding of the Ras Activator Son of Sevenless to Insulin Receptor Substrate-1 Signaling Complexes
5. BARBACID, M, RAS GENES, ANNUAL REVIEW OF BIOCHEMISTRY 56: 779 (1987).
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