Neural Mechanisms of a Genome-Wide Supported Psychosis Variant-Reference-Cited by-同舟云学术

Neural Mechanisms of a Genome-Wide Supported Psychosis Variant

Published:2009-05 Issue:5927 Volume:324 Page:605-605
ISSN:0036-8075
Container-title:Science
language:en
Short-container-title:Science

Author:

Esslinger Christine¹,Walter Henrik²³,Kirsch Peter¹,Erk Susanne³,Schnell Knut²³,Arnold Claudia³,Haddad Leila¹,Mier Daniela¹,Opitz von Boberfeld Carola³,Raab Kyeon¹,Witt Stephanie H.⁴,Rietschel Marcella⁴,Cichon Sven⁵,Meyer-Lindenberg Andreas¹

Affiliation:

1. Department of Psychiatry and Psychotherapy, Central Institute of Mental Health, University of Heidelberg, J5, 68159 Mannheim, Germany.

2. Division of Medical Psychology, University of Bonn, 53105 Bonn, Germany.

3. Department of Psychiatry and Psychotherapy, University of Bonn, 53105 Bonn, Germany.

4. Department of Genetic Epidemiology in Psychiatry, Central Institute of Mental Health, University of Heidelberg, J5, 68159 Mannheim, Germany.

5. Department of Genomics, Life and Brain Center and Institute of Human Genetics, University of Bonn, 53105 Bonn, Germany.

Abstract

A genetic polymorphism associated with schizophrenia conveys a risk for abnormal connectivity between brain regions.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference6 articles.

1. Synaptic Plasticity and Dysconnection in Schizophrenia

2. Regionally Specific Disturbance of Dorsolateral Prefrontal–Hippocampal Functional Connectivity in Schizophrenia

3. Identification of loci associated with schizophrenia by genome-wide association and follow-up

4. Intermediate phenotypes and genetic mechanisms of psychiatric disorders

5. Evidence That Altered Amygdala Activity in Schizophrenia Is Related to Clinical State and Not Genetic Risk

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