β-Adrenergic Receptor Kinase-2 and β-Arrestin-2 as Mediators of Odorant-Induced Desensitization-Reference-Cited by-同舟云学术

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β-Adrenergic Receptor Kinase-2 and β-Arrestin-2 as Mediators of Odorant-Induced Desensitization

Published:1993-02-05 Issue:5096 Volume:259 Page:825-829
ISSN:0036-8075
Container-title:Science
language:en
Short-container-title:Science

Author:

Dawson Ted M.¹,Arriza Jeffrey L.²,Jaworsky Donna E.¹,Borisy Felice F.¹,Attramadal Håvard²,Lefkowitz Robert J.²,Ronnett Gabriele V.¹

Affiliation:

1. Departments of Neuroscience and Neurology, The Johns Hopkins Medical Institutions, 725 North Wolfe Street, Baltimore, MD 21205.

2. Howard Hughes Medical Institute, Department of Medicine and Biochemistry, Duke University Medical Center, Box 3821, Durham, NC 27710.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference33 articles.

1. The G-protein-coupled receptor kinases beta ARK1 and beta ARK2 are widely distributed at synapses in rat brain

2. Beta-arrestin2, a novel member of the arrestin/beta-arrestin gene family.

3. Identification of a Specialized Adenylyl Cyclase That May Mediate Odorant Detection

4. Regulation of Adenylyl Cyclase-Coupled beta-Adrenergic Receptors

5. Cloning, expression, and chromosomal localization of beta-adrenergic receptor kinase 2. A new member of the receptor kinase family

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