Neuromuscular Junction in Myasthenia Gravis: Decreased Acetylcholine Receptors
Author:
Affiliation:
1. Department of Embryology, Carnegie Institution of Washington, Baltimore, Maryland 21210
2. Johns Hopkins University School of Medicine, Baltimore, Maryland 21205
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Reference19 articles.
1. BARNARD, E.A., CHOLINERGIC RECEPTOR MOLECULES AND CHOLINESTERASE MOLECULES AT MOUSE SKELETAL MUSCLE JUNCTIONS, NATURE 234: 207 (1971).
2. BERG, D.K., BINDING OF ALPHA-BUNGAROTOXIN TO ACETYLCHOLINE RECEPTORS IN MAMMALIAN MUSCLE, PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 69: 147 (1972).
3. CHANG, C.C., N,O-DI AND N,N,O-TRI [H-3] ACETYL ALPHA-BUNGAROTOXINS AS SPECIFIC LABELING AGENTS OF CHOLINERGIC RECEPTORS, BRITISH JOURNAL OF PHARMACOLOGY 47: 147 (1973).
4. CHANG, C.C., INFLUENCE OF CHRONIC NEOSTIGMINE TREATMENT ON NUMBER OF ACETYLCHOLINE RECEPTORS AND RELEASE OF ACETYLCHOLINE FROM RAT DIAPHRAGM, JOURNAL OF PHYSIOLOGY-LONDON 230: 613 (1973).
5. CHANGEUX, J.P., USE OF A SNAKE VENON TOXIN TO CHARACTERIZE CHOLINERGIC RECEPTOR PROTEIN, PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 67: 1241 (1970).
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