Abnormal Centrosome Amplification in the Absence of p53

Author:

Fukasawa Kenji1,Choi Taesaeng1,Kuriyama Ryoko2,Rulong Shen1,Woude George F. Vande1

Affiliation:

1. K. Fukasawa, T. Choi, S. Rulong, G. F. Vande Woude, ABL-Basic Research Program, National Cancer Institute, Frederick Cancer Research and Development Center, Post Office Box B, Frederick, MD 21702-1201, USA.

2. R. Kuriyama, Department of Cell Biology and Neuroanatomy, University of Minnesota Medical School, Minneapolis, MN 55455, USA.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference57 articles.

1. P53 CONTROLS BOTH THE G(2)/M AND THE G(1) CELL-CYCLE CHECKPOINTS AND MEDIATES REVERSIBLE GROWTH ARREST IN HUMAN FIBROBLASTS;AGARWAL M.L.;PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA,1995

2. DISSOCIATION OF CENTROSOME REPLICATION EVENTS FROM CYCLES OF DNA-SYNTHESIS AND MITOTIC DIVISION IN HYDROXYUREA-ARRESTED CHINESE-HAMSTER OVARY CELLS;BALCZON R;JOURNAL OF CELL BIOLOGY,1995

3. FRIEND VIRUS-INDUCED ERYTHROLEUKEMIA AND THE MULTISTAGE NATURE OF CANCER;BENDAVID Y;CELL,1991

4. BLAIRZAJDEL M.E., ONCOGENE 2, 579 (1988).

5. BOMENS M, CENTROSOME: 1 (1992).

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