Physiologic Implications of Phosphoinositides and Phospholipase C in the Regulation of Insulin Secretion-Reference-Cited by-同舟云学术

Physiologic Implications of Phosphoinositides and Phospholipase C in the Regulation of Insulin Secretion

Published:2010 Issue:1 Volume:56 Page:1-8
ISSN:0301-4800
Container-title:Journal of Nutritional Science and Vitaminology
language:en
Short-container-title:J Nutr Sci Vitaminol

Author:

YAMAZAKI Hanae¹,ZAWALICH Kathleen C.²,ZAWALICH Walter S.²

Affiliation:

1. Laboratory of Ajinomoto Integrative Research for Advanced Dieting, Graduate School of Agriculture, Kyoto University

2. Yale University School of Nursing

Publisher

Center for Academic Publications Japan

Subject

Nutrition and Dietetics,Medicine (miscellaneous)

Link

https://www.jstage.jst.go.jp/article/jnsv/56/1/56_1_1/_pdf

Reference72 articles.

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3. 3) Zawalich WS. 1988. Modulation of insulin secretion from beta-cells by phosphoinositide-derived second-messenger molecules. Diabetes 37: 137-141.

4. 4) Eto K, Tsubamoto Y, Terauchi Y, Waki K, Kubota N, Taka J, Tamemoto H, Tobe K, Noda M, Kadowaki T. 2000. Role of IRS-1/2 and PI-3 kinase pathway in the regulation of β-cell mass and glucose-stimulated insulin secretion. Diabetes 49: A45 (abstract).

5. 5) Eto K, Yamashita T, Tsubamoto Y, Terauchi Y, Hirose K, Kubota N, Yamashita S, Taka J, Satoh S, Sekihara H, Tobe K, Iino M, Noda M, Kimura S, Kadowaki T. 2002. Phosphatidylinositol 3-kinase suppresses glucose-stimulated insulin secretion by affecting post-cytosolic [Ca²⁺] elevation signals. Diabetes 51: 87-97.

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